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The neuronal and immune memory systems as supervisors of neural plasticity and aging of the brain - From phenomenology to coding of information
(New York Acad Sciences, New York, 2005)
The ultimate goal of this report is to learn how to manipulate the level of memory T cells for more effective treatment of such neurological diseases as multiple sclerosis (MS), where certain T cell subsets recognize ...
Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease
(IEEE, New York, 2002)
In a previous paper, the authors have described the effect of Ca2+-antagonist. verapamil on Na,K-ATPase in experimental model of Alzheimer's disease (AD-[38,39]. The present paper is concerned with the effect of magnetic, ...
Comparison of the value of T3, T4 and TSH in sera of animals treated with low and high dose concentrated potassium iodine solutions / Poređenje nivoa hormona T3, T4 i TSH u serumu životinja tretiranih niskom i visokom koncentracijom rastvora kalijum jodida
(Univerzitet u Beogradu - Medicinski fakultet, Beograd, 2004)
Etiopathogenesis of the most frequent endocrinopathy, Hashimoto thyroiditis (HT) is not clear. Our investigations dating from 1990 on an experimental model for examination of thyroiditis in Wistar rats show morphological ...
Ethanol consumption affecting some brain proteins and cortical plasticity might disturb the neural networks of long-term memory
(IEEE, New York, 2004)
The objective of this study is to investigate whether chronic ethanol consumption include brain damage expressed as; an increase of antibody titer to S-100 and synaptic membrane proteins, additioning of lateral ventricles ...
Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat
(IOS Press, Amsterdam, 2008)
Several studies suggested that the activity of erythrocyte Na, K-ATPase declines with aging. Here, it is postulated that alterations in the substrate kinetics of the erythrocyte membrane Na, K-ATPase could be more aggravated ...
Magnetic brain stimulation upregulates adhesion and prevents EAE: MMP-2, ICAM-1, and VCAM-1 in the choroid plexus as a target
(Taylor & Francis Ltd, Abingdon, 2009)
We are most thankful to Prof. Yehuda Shoenfeld, head of Autoimmunity Center, Sheba Hospital at Tel Aviv, University in Israel, for critical and constructive comments on the manuscript. Thanks to Prof. B. Reljin and N. ...
Effect of neuropeptide Y on inflammatory paw edema in the rat: involvement of peripheral NPYY1 and Y5 receptors and interaction with dipeptidyl-peptidase IV (CD26)
(Elsevier, Amsterdam, 2002)
Several lines of evidence suggest that neuropeptide Y (NPY) may exert regulatory effects in local inflammatory responses. Here, we show that intraplantarly (i.pl.) applied NPY, peptide YY (PYY), and an NPY Y5 receptor-selective ...