TY  - JOUR
AU  - Erić-Jovičić, Milena
AU  - Popović, Miroljub
AU  - Jovanova-Nešić, Katica
AU  - Popović, Natalija
AU  - Jovičić-Pavlović, Svetlana
AU  - Rakić, Ljubisav
PY  - 2008
UR  - http://intor.torlakinstitut.com/handle/123456789/247
AB  - Several studies suggested that the activity of erythrocyte Na, K-ATPase declines with aging. Here, it is postulated that alterations in the substrate kinetics of the erythrocyte membrane Na, K-ATPase could be more aggravated in conditions of brain cholinergic dysfunction seen in Alzheimer's disease than in normal aging. To test this hypothesis, we compared the Na, K-ATPase activity (Vmax/Km parameters) in aged rats with those in young rats with brain cholinergic dysfunction induced by electrolytic-, kainic acid-lesioned nucleus basalis magnocellularis (NBM) or by intracerebroventricular AlCl3 administration. In the above mentioned groups, Vmax values were significantly lower in comparison to the control animals. Furthermore, Km values were significantly higher in animals with electrolytic-induced NBM lesions, AlCl3 treated rats and aged animals. However, Km was significantly lower in kainic acid-induced NBM lesions compared to the control group. The Na, K-ATPase catalytic efficiency, estimated by the ratio Vm/Km, decreased as followed: young animals  gt  aged animals  gt  kainic acid lesion  gt  electrolityc lesion  gt  AlCl3. Our data suggest that neurodegenerative processes similar to those seen in Alzheimer's disease affect the sodium/potassium pump functionality which might be detected in peripheral blood erythrocyte membranes.
PB  - IOS Press, Amsterdam
T2  - Journal of Alzheimer's Disease
T1  - Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat
EP  - 93
IS  - 1
SP  - 85
VL  - 14
DO  - 10.3233/JAD-2008-14108
ER  - 

@article{
author = "Erić-Jovičić, Milena and Popović, Miroljub and Jovanova-Nešić, Katica and Popović, Natalija and Jovičić-Pavlović, Svetlana and Rakić, Ljubisav",
year = "2008",
abstract = "Several studies suggested that the activity of erythrocyte Na, K-ATPase declines with aging. Here, it is postulated that alterations in the substrate kinetics of the erythrocyte membrane Na, K-ATPase could be more aggravated in conditions of brain cholinergic dysfunction seen in Alzheimer's disease than in normal aging. To test this hypothesis, we compared the Na, K-ATPase activity (Vmax/Km parameters) in aged rats with those in young rats with brain cholinergic dysfunction induced by electrolytic-, kainic acid-lesioned nucleus basalis magnocellularis (NBM) or by intracerebroventricular AlCl3 administration. In the above mentioned groups, Vmax values were significantly lower in comparison to the control animals. Furthermore, Km values were significantly higher in animals with electrolytic-induced NBM lesions, AlCl3 treated rats and aged animals. However, Km was significantly lower in kainic acid-induced NBM lesions compared to the control group. The Na, K-ATPase catalytic efficiency, estimated by the ratio Vm/Km, decreased as followed: young animals  gt  aged animals  gt  kainic acid lesion  gt  electrolityc lesion  gt  AlCl3. Our data suggest that neurodegenerative processes similar to those seen in Alzheimer's disease affect the sodium/potassium pump functionality which might be detected in peripheral blood erythrocyte membranes.",
publisher = "IOS Press, Amsterdam",
journal = "Journal of Alzheimer's Disease",
title = "Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat",
pages = "93-85",
number = "1",
volume = "14",
doi = "10.3233/JAD-2008-14108"
}

Erić-Jovičić, M., Popović, M., Jovanova-Nešić, K., Popović, N., Jovičić-Pavlović, S.,& Rakić, L.. (2008). Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat. in Journal of Alzheimer's Disease
IOS Press, Amsterdam., 14(1), 85-93.
https://doi.org/10.3233/JAD-2008-14108

Erić-Jovičić M, Popović M, Jovanova-Nešić K, Popović N, Jovičić-Pavlović S, Rakić L. Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat. in Journal of Alzheimer's Disease. 2008;14(1):85-93.
doi:10.3233/JAD-2008-14108 .

Erić-Jovičić, Milena, Popović, Miroljub, Jovanova-Nešić, Katica, Popović, Natalija, Jovičić-Pavlović, Svetlana, Rakić, Ljubisav, "Aging, aluminium and basal forebrain lesions modify substrate kinetics of erythrocyte membrane Na, K-ATPase in the rat" in Journal of Alzheimer's Disease, 14, no. 1 (2008):85-93,
https://doi.org/10.3233/JAD-2008-14108 . .

TY  - CONF
AU  - Erić-Jovičić, Milena
AU  - Jovičić-Pavlović, Svetlana
PY  - 2008
UR  - http://intor.torlakinstitut.com/handle/123456789/265
PB  - Wiley-Blackwell, Malden
C3  - European Journal of Neurology
T1  - Scoring according to the Hamilton and Zung scales for the rating of depression in patients suffering from Parkinson's disease
EP  - 108
SP  - 108
VL  - 15
UR  - https://hdl.handle.net/21.15107/rcub_intor_265
ER  - 

@conference{
author = "Erić-Jovičić, Milena and Jovičić-Pavlović, Svetlana",
year = "2008",
publisher = "Wiley-Blackwell, Malden",
journal = "European Journal of Neurology",
title = "Scoring according to the Hamilton and Zung scales for the rating of depression in patients suffering from Parkinson's disease",
pages = "108-108",
volume = "15",
url = "https://hdl.handle.net/21.15107/rcub_intor_265"
}

Erić-Jovičić, M.,& Jovičić-Pavlović, S.. (2008). Scoring according to the Hamilton and Zung scales for the rating of depression in patients suffering from Parkinson's disease. in European Journal of Neurology
Wiley-Blackwell, Malden., 15, 108-108.
https://hdl.handle.net/21.15107/rcub_intor_265

Erić-Jovičić M, Jovičić-Pavlović S. Scoring according to the Hamilton and Zung scales for the rating of depression in patients suffering from Parkinson's disease. in European Journal of Neurology. 2008;15:108-108.
https://hdl.handle.net/21.15107/rcub_intor_265 .

Erić-Jovičić, Milena, Jovičić-Pavlović, Svetlana, "Scoring according to the Hamilton and Zung scales for the rating of depression in patients suffering from Parkinson's disease" in European Journal of Neurology, 15 (2008):108-108,
https://hdl.handle.net/21.15107/rcub_intor_265 .

TY  - CONF
AU  - Erić-Jovičić, Milena
AU  - Jovičić-Pavlović, Svetlana
PY  - 2008
UR  - http://intor.torlakinstitut.com/handle/123456789/263
PB  - Wiley-Blackwell, Malden
C3  - European Journal of Neurology
T1  - Scoring according to the MMS scale for the rating of dementia in patients suffering from Parkinson's disease in relation to the length of treatment
EP  - 38
SP  - 38
VL  - 15
UR  - https://hdl.handle.net/21.15107/rcub_intor_263
ER  - 

@conference{
author = "Erić-Jovičić, Milena and Jovičić-Pavlović, Svetlana",
year = "2008",
publisher = "Wiley-Blackwell, Malden",
journal = "European Journal of Neurology",
title = "Scoring according to the MMS scale for the rating of dementia in patients suffering from Parkinson's disease in relation to the length of treatment",
pages = "38-38",
volume = "15",
url = "https://hdl.handle.net/21.15107/rcub_intor_263"
}

Erić-Jovičić, M.,& Jovičić-Pavlović, S.. (2008). Scoring according to the MMS scale for the rating of dementia in patients suffering from Parkinson's disease in relation to the length of treatment. in European Journal of Neurology
Wiley-Blackwell, Malden., 15, 38-38.
https://hdl.handle.net/21.15107/rcub_intor_263

Erić-Jovičić M, Jovičić-Pavlović S. Scoring according to the MMS scale for the rating of dementia in patients suffering from Parkinson's disease in relation to the length of treatment. in European Journal of Neurology. 2008;15:38-38.
https://hdl.handle.net/21.15107/rcub_intor_263 .

Erić-Jovičić, Milena, Jovičić-Pavlović, Svetlana, "Scoring according to the MMS scale for the rating of dementia in patients suffering from Parkinson's disease in relation to the length of treatment" in European Journal of Neurology, 15 (2008):38-38,
https://hdl.handle.net/21.15107/rcub_intor_263 .

TY  - JOUR
AU  - Jovanova-Nešić, Katica
AU  - Erić-Jovičić, Milena
AU  - Spector, Novera Herbert
PY  - 2006
UR  - http://intor.torlakinstitut.com/handle/123456789/207
AB  - This article reports here on the influence of the static magnetic fields ( MFs), locally applied to the brain area, on Na, K-ATPase activity in the rat with lesioned nucleus basalis magnocellularis ( NBM) by intracerebral injection of 5 mu l, 1% AlCl3 into the nucleus. Two AKMA micromagnets ( M) flux density of 60 miliTesla, 5 mm in diameter, were bilaterally implanted with "N" polarity facing down to the cranial bones in the vicinity of the pineal gland ( PG), immediately after the lesioning of NBM, during the same operation procedure. Ten days after the lesions of NBM, Na, K-ATPase activity on the erythrocyte membranes in the peripheral blood, measured spectrophotometrically, was completely inhibited. Magnetic stimulation ( 60 mT) of the brain during the 10 days significantly increased Na, K-ATPase activity on the erythrocyte membranes of rats with lesioned NBM. This results suggests that altered by lesions Na, K-ATPase activity in an experimental model of Alzheimer's disease might be ameliorated by magnetic stimulation of the brain.
PB  - Taylor & Francis Ltd, Abingdon
T2  - International Journal of Neuroscience
T1  - Magnetic stimulation of the brain increase Na+, K+-atpase activity decreased by injection of alcl(3) into nucleus basalis magnocellularis of rats
EP  - 695
IS  - 6
SP  - 681
VL  - 116
DO  - 10.1080/00207450600674830
ER  - 

@article{
author = "Jovanova-Nešić, Katica and Erić-Jovičić, Milena and Spector, Novera Herbert",
year = "2006",
abstract = "This article reports here on the influence of the static magnetic fields ( MFs), locally applied to the brain area, on Na, K-ATPase activity in the rat with lesioned nucleus basalis magnocellularis ( NBM) by intracerebral injection of 5 mu l, 1% AlCl3 into the nucleus. Two AKMA micromagnets ( M) flux density of 60 miliTesla, 5 mm in diameter, were bilaterally implanted with "N" polarity facing down to the cranial bones in the vicinity of the pineal gland ( PG), immediately after the lesioning of NBM, during the same operation procedure. Ten days after the lesions of NBM, Na, K-ATPase activity on the erythrocyte membranes in the peripheral blood, measured spectrophotometrically, was completely inhibited. Magnetic stimulation ( 60 mT) of the brain during the 10 days significantly increased Na, K-ATPase activity on the erythrocyte membranes of rats with lesioned NBM. This results suggests that altered by lesions Na, K-ATPase activity in an experimental model of Alzheimer's disease might be ameliorated by magnetic stimulation of the brain.",
publisher = "Taylor & Francis Ltd, Abingdon",
journal = "International Journal of Neuroscience",
title = "Magnetic stimulation of the brain increase Na+, K+-atpase activity decreased by injection of alcl(3) into nucleus basalis magnocellularis of rats",
pages = "695-681",
number = "6",
volume = "116",
doi = "10.1080/00207450600674830"
}

Jovanova-Nešić, K., Erić-Jovičić, M.,& Spector, N. H.. (2006). Magnetic stimulation of the brain increase Na+, K+-atpase activity decreased by injection of alcl(3) into nucleus basalis magnocellularis of rats. in International Journal of Neuroscience
Taylor & Francis Ltd, Abingdon., 116(6), 681-695.
https://doi.org/10.1080/00207450600674830

Jovanova-Nešić K, Erić-Jovičić M, Spector NH. Magnetic stimulation of the brain increase Na+, K+-atpase activity decreased by injection of alcl(3) into nucleus basalis magnocellularis of rats. in International Journal of Neuroscience. 2006;116(6):681-695.
doi:10.1080/00207450600674830 .

Jovanova-Nešić, Katica, Erić-Jovičić, Milena, Spector, Novera Herbert, "Magnetic stimulation of the brain increase Na+, K+-atpase activity decreased by injection of alcl(3) into nucleus basalis magnocellularis of rats" in International Journal of Neuroscience, 116, no. 6 (2006):681-695,
https://doi.org/10.1080/00207450600674830 . .

TY  - CONF
AU  - Jovanova-Nešić, Katica
AU  - Erić-Jovičić, Milena
AU  - Popović, M.
AU  - Popović, N.
AU  - Rakić, L.
AU  - Spector, Novera Herbert
PY  - 2002
UR  - http://intor.torlakinstitut.com/handle/123456789/149
AB  - In a previous paper, the authors have described the effect of Ca2+-antagonist. verapamil on Na,K-ATPase in experimental model of Alzheimer's disease (AD-[38,39]. The present paper is concerned with the effect of magnetic, stimulation of. pineal complex on Na,K-ATPase activity in the same experimental model of AD. Because accumulating data indicate that free radicals mediate injury and death of neurons in AD, and because magnetic fields (MFs) can alter- free radicals. reactions; we tested the hypothesis that stationary MFs mediates ion homeostasis through membrane Na;K-ATPase activity. Results are presented as Vmax/Km - parameters on erythrocyte membranes in peripheral blood of rats with lesioned nucleus basalis magnocellularis. Bilateral electrolytic or by kainic acid induced lesions of NBM induce significant decrease of Vmax/Km activity on erythrocyte membranes obtained by cardiac punction. Stimulation of - pineal complex of the brain more than ten days, by magnetic beards (600-Gauss flux density), fixed on the skull upon pineal gland, significantly increase impaired by lesions of NBM, Na, K-ATPase activity. Results are presented as Vmax/Km parameters. on erythrocyte membranes in peripheral blood of rats with lesioned NBM of the basal forebrain bundle. Chronically magnetic stimulation of the pineal complex significantly increase maximum velocity (Vmax; nmol Pi/mg protein/min) of proteins in both lesioned group, even more than 2-fold in by kainic acid (ka)lesioned animals in comparison of lesioned sham-stimulated and increase Vmax in comparison to both controls (sham-lesioned m-sham-stimulated, and intact controls, and return desturbed by lesions affinity of enzyme to substrate (Km; nM) near to the control values. These results confirm the hypothesis that altered ion homeostasis disturbed by neurodegenerations play an essential role in pathogenesis of experimental Alzheimer's disease (AD) and that magnetic stimulation of the pineal complex might successfully restore disturbed by neuronal death Na, K-ATPase activity.
PB  - IEEE, New York
C3  - 2002 6th Seminar on Neural Network Applications in Electrical Engineering, NEUREL 2002 - Proceedings
T1  - Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease
EP  - 170
SP  - 165
UR  - https://hdl.handle.net/21.15107/rcub_intor_149
ER  - 

@conference{
author = "Jovanova-Nešić, Katica and Erić-Jovičić, Milena and Popović, M. and Popović, N. and Rakić, L. and Spector, Novera Herbert",
year = "2002",
abstract = "In a previous paper, the authors have described the effect of Ca2+-antagonist. verapamil on Na,K-ATPase in experimental model of Alzheimer's disease (AD-[38,39]. The present paper is concerned with the effect of magnetic, stimulation of. pineal complex on Na,K-ATPase activity in the same experimental model of AD. Because accumulating data indicate that free radicals mediate injury and death of neurons in AD, and because magnetic fields (MFs) can alter- free radicals. reactions; we tested the hypothesis that stationary MFs mediates ion homeostasis through membrane Na;K-ATPase activity. Results are presented as Vmax/Km - parameters on erythrocyte membranes in peripheral blood of rats with lesioned nucleus basalis magnocellularis. Bilateral electrolytic or by kainic acid induced lesions of NBM induce significant decrease of Vmax/Km activity on erythrocyte membranes obtained by cardiac punction. Stimulation of - pineal complex of the brain more than ten days, by magnetic beards (600-Gauss flux density), fixed on the skull upon pineal gland, significantly increase impaired by lesions of NBM, Na, K-ATPase activity. Results are presented as Vmax/Km parameters. on erythrocyte membranes in peripheral blood of rats with lesioned NBM of the basal forebrain bundle. Chronically magnetic stimulation of the pineal complex significantly increase maximum velocity (Vmax; nmol Pi/mg protein/min) of proteins in both lesioned group, even more than 2-fold in by kainic acid (ka)lesioned animals in comparison of lesioned sham-stimulated and increase Vmax in comparison to both controls (sham-lesioned m-sham-stimulated, and intact controls, and return desturbed by lesions affinity of enzyme to substrate (Km; nM) near to the control values. These results confirm the hypothesis that altered ion homeostasis disturbed by neurodegenerations play an essential role in pathogenesis of experimental Alzheimer's disease (AD) and that magnetic stimulation of the pineal complex might successfully restore disturbed by neuronal death Na, K-ATPase activity.",
publisher = "IEEE, New York",
journal = "2002 6th Seminar on Neural Network Applications in Electrical Engineering, NEUREL 2002 - Proceedings",
title = "Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease",
pages = "170-165",
url = "https://hdl.handle.net/21.15107/rcub_intor_149"
}

Jovanova-Nešić, K., Erić-Jovičić, M., Popović, M., Popović, N., Rakić, L.,& Spector, N. H.. (2002). Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease. in 2002 6th Seminar on Neural Network Applications in Electrical Engineering, NEUREL 2002 - Proceedings
IEEE, New York., 165-170.
https://hdl.handle.net/21.15107/rcub_intor_149

Jovanova-Nešić K, Erić-Jovičić M, Popović M, Popović N, Rakić L, Spector NH. Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease. in 2002 6th Seminar on Neural Network Applications in Electrical Engineering, NEUREL 2002 - Proceedings. 2002;:165-170.
https://hdl.handle.net/21.15107/rcub_intor_149 .

Jovanova-Nešić, Katica, Erić-Jovičić, Milena, Popović, M., Popović, N., Rakić, L., Spector, Novera Herbert, "Effect of magnetic stimulation of pineal complex of the brain on Na,K-ATPase in experimental Alzheimer's Disease" in 2002 6th Seminar on Neural Network Applications in Electrical Engineering, NEUREL 2002 - Proceedings (2002):165-170,
https://hdl.handle.net/21.15107/rcub_intor_149 .

TY  - JOUR
AU  - Popović, M.
AU  - Popović, N.
AU  - Erić-Jovičić, Milena
AU  - Jovanova-Nešić, Katica
PY  - 2000
UR  - http://intor.torlakinstitut.com/handle/123456789/122
AB  - The present study was designed to establish the influence of chronic social isolation stress on humoral and cellular immunity in nucleus basalis magnocellularis (NBM)-lesioned rats. Therefore, ten days after bilateral electrolytic lesions of NBM, adult male Wistar rats were immunized with bovine serum albumin in complete Freund's adjuvant (BSA-CFA) and placed individually or in groups of five rats during 21 days. On days 10 and 21 after immunization, the Arthus and delayed hypersensitivity skin reactions to BSA as well as anti-BSA antibody production were determined. On day 10, the diameter and intensity of delayed hypersensitivity skin reaction to BSA were significantly higher in social-isolated rats in comparison with the group-reared ones. On day 21, the diameter and intensity of the Arthus skin reaction were significantly higher in social-isolated rats compared to group-reared rats. Between days 10 and 21, the diameter and intensity of the Arthus skin reaction significantly increased in social-isolated rats, while the diameter of delayed hypersensitivity skin reaction significantly decreased. In contrast to social-isolated rats, there were no significant differences in Arthus and delayed hypersensitivity skin reactions in group-reared rats, between days 10 and 21. Also there were no significant differences in the production of anti-BSA antibody between social-isolated and group-reared rats. The relative spleen weight was significantly lower in social-isolated rats. These data suggest that chronic isolation stress modify humoral and cellular immunity in NBM-lesioned rats.
PB  - Taylor & Francis Ltd, Abingdon
T2  - International Journal of Neuroscience
T1  - Immune responses in nucleus basalis magnocellularis-lesioned rats exposed to chronic isolation stress
EP  - 131
IS  - 1-4
SP  - 125
VL  - 100
DO  - 10.3109/00207450008999683
ER  - 

@article{
author = "Popović, M. and Popović, N. and Erić-Jovičić, Milena and Jovanova-Nešić, Katica",
year = "2000",
abstract = "The present study was designed to establish the influence of chronic social isolation stress on humoral and cellular immunity in nucleus basalis magnocellularis (NBM)-lesioned rats. Therefore, ten days after bilateral electrolytic lesions of NBM, adult male Wistar rats were immunized with bovine serum albumin in complete Freund's adjuvant (BSA-CFA) and placed individually or in groups of five rats during 21 days. On days 10 and 21 after immunization, the Arthus and delayed hypersensitivity skin reactions to BSA as well as anti-BSA antibody production were determined. On day 10, the diameter and intensity of delayed hypersensitivity skin reaction to BSA were significantly higher in social-isolated rats in comparison with the group-reared ones. On day 21, the diameter and intensity of the Arthus skin reaction were significantly higher in social-isolated rats compared to group-reared rats. Between days 10 and 21, the diameter and intensity of the Arthus skin reaction significantly increased in social-isolated rats, while the diameter of delayed hypersensitivity skin reaction significantly decreased. In contrast to social-isolated rats, there were no significant differences in Arthus and delayed hypersensitivity skin reactions in group-reared rats, between days 10 and 21. Also there were no significant differences in the production of anti-BSA antibody between social-isolated and group-reared rats. The relative spleen weight was significantly lower in social-isolated rats. These data suggest that chronic isolation stress modify humoral and cellular immunity in NBM-lesioned rats.",
publisher = "Taylor & Francis Ltd, Abingdon",
journal = "International Journal of Neuroscience",
title = "Immune responses in nucleus basalis magnocellularis-lesioned rats exposed to chronic isolation stress",
pages = "131-125",
number = "1-4",
volume = "100",
doi = "10.3109/00207450008999683"
}

Popović, M., Popović, N., Erić-Jovičić, M.,& Jovanova-Nešić, K.. (2000). Immune responses in nucleus basalis magnocellularis-lesioned rats exposed to chronic isolation stress. in International Journal of Neuroscience
Taylor & Francis Ltd, Abingdon., 100(1-4), 125-131.
https://doi.org/10.3109/00207450008999683

Popović M, Popović N, Erić-Jovičić M, Jovanova-Nešić K. Immune responses in nucleus basalis magnocellularis-lesioned rats exposed to chronic isolation stress. in International Journal of Neuroscience. 2000;100(1-4):125-131.
doi:10.3109/00207450008999683 .

Popović, M., Popović, N., Erić-Jovičić, Milena, Jovanova-Nešić, Katica, "Immune responses in nucleus basalis magnocellularis-lesioned rats exposed to chronic isolation stress" in International Journal of Neuroscience, 100, no. 1-4 (2000):125-131,
https://doi.org/10.3109/00207450008999683 . .

TY  - CONF
AU  - Popović, M.
AU  - Popović, N.
AU  - Erić-Jovičić, Milena
AU  - Inić, Aleksandra
PY  - 1998
UR  - http://intor.torlakinstitut.com/handle/123456789/100
PB  - Wiley, Hoboken
C3  - European Journal of Neuroscience
T1  - Effect of physostigmine on cold restraint-induced gastric lesions in an experimental, model of Alzheimer's disease
EP  - 94
SP  - 94
VL  - 10
UR  - https://hdl.handle.net/21.15107/rcub_intor_100
ER  - 

@conference{
author = "Popović, M. and Popović, N. and Erić-Jovičić, Milena and Inić, Aleksandra",
year = "1998",
publisher = "Wiley, Hoboken",
journal = "European Journal of Neuroscience",
title = "Effect of physostigmine on cold restraint-induced gastric lesions in an experimental, model of Alzheimer's disease",
pages = "94-94",
volume = "10",
url = "https://hdl.handle.net/21.15107/rcub_intor_100"
}

Popović, M., Popović, N., Erić-Jovičić, M.,& Inić, A.. (1998). Effect of physostigmine on cold restraint-induced gastric lesions in an experimental, model of Alzheimer's disease. in European Journal of Neuroscience
Wiley, Hoboken., 10, 94-94.
https://hdl.handle.net/21.15107/rcub_intor_100

Popović M, Popović N, Erić-Jovičić M, Inić A. Effect of physostigmine on cold restraint-induced gastric lesions in an experimental, model of Alzheimer's disease. in European Journal of Neuroscience. 1998;10:94-94.
https://hdl.handle.net/21.15107/rcub_intor_100 .

Popović, M., Popović, N., Erić-Jovičić, Milena, Inić, Aleksandra, "Effect of physostigmine on cold restraint-induced gastric lesions in an experimental, model of Alzheimer's disease" in European Journal of Neuroscience, 10 (1998):94-94,
https://hdl.handle.net/21.15107/rcub_intor_100 .

TY  - CONF
AU  - Popović, N.
AU  - Erić-Jovičić, Milena
AU  - Petrićević, Marijana
AU  - Popović, M.
PY  - 1998
UR  - http://intor.torlakinstitut.com/handle/123456789/99
PB  - Wiley, Hoboken
C3  - European Journal of Neuroscience
T1  - Thermoregulation in an animal model of Alzheimer's disease
EP  - 94
SP  - 94
VL  - 10
UR  - https://hdl.handle.net/21.15107/rcub_intor_99
ER  - 

@conference{
author = "Popović, N. and Erić-Jovičić, Milena and Petrićević, Marijana and Popović, M.",
year = "1998",
publisher = "Wiley, Hoboken",
journal = "European Journal of Neuroscience",
title = "Thermoregulation in an animal model of Alzheimer's disease",
pages = "94-94",
volume = "10",
url = "https://hdl.handle.net/21.15107/rcub_intor_99"
}

Popović, N., Erić-Jovičić, M., Petrićević, M.,& Popović, M.. (1998). Thermoregulation in an animal model of Alzheimer's disease. in European Journal of Neuroscience
Wiley, Hoboken., 10, 94-94.
https://hdl.handle.net/21.15107/rcub_intor_99

Popović N, Erić-Jovičić M, Petrićević M, Popović M. Thermoregulation in an animal model of Alzheimer's disease. in European Journal of Neuroscience. 1998;10:94-94.
https://hdl.handle.net/21.15107/rcub_intor_99 .

Popović, N., Erić-Jovičić, Milena, Petrićević, Marijana, Popović, M., "Thermoregulation in an animal model of Alzheimer's disease" in European Journal of Neuroscience, 10 (1998):94-94,
https://hdl.handle.net/21.15107/rcub_intor_99 .

TY  - CONF
AU  - Popović, N.
AU  - Erić-Jovičić, Milena
AU  - Popović, M.
PY  - 1998
UR  - http://intor.torlakinstitut.com/handle/123456789/90
PB  - Wiley, Hoboken
C3  - European Journal of Neuroscience
T1  - Pharmacological and transplantational approaches to treatment of learning and memory impairment in an animal model of Alzheimer's disease
EP  - 437
SP  - 437
VL  - 10
UR  - https://hdl.handle.net/21.15107/rcub_intor_90
ER  - 

@conference{
author = "Popović, N. and Erić-Jovičić, Milena and Popović, M.",
year = "1998",
publisher = "Wiley, Hoboken",
journal = "European Journal of Neuroscience",
title = "Pharmacological and transplantational approaches to treatment of learning and memory impairment in an animal model of Alzheimer's disease",
pages = "437-437",
volume = "10",
url = "https://hdl.handle.net/21.15107/rcub_intor_90"
}

Popović, N., Erić-Jovičić, M.,& Popović, M.. (1998). Pharmacological and transplantational approaches to treatment of learning and memory impairment in an animal model of Alzheimer's disease. in European Journal of Neuroscience
Wiley, Hoboken., 10, 437-437.
https://hdl.handle.net/21.15107/rcub_intor_90

Popović N, Erić-Jovičić M, Popović M. Pharmacological and transplantational approaches to treatment of learning and memory impairment in an animal model of Alzheimer's disease. in European Journal of Neuroscience. 1998;10:437-437.
https://hdl.handle.net/21.15107/rcub_intor_90 .

Popović, N., Erić-Jovičić, Milena, Popović, M., "Pharmacological and transplantational approaches to treatment of learning and memory impairment in an animal model of Alzheimer's disease" in European Journal of Neuroscience, 10 (1998):437-437,
https://hdl.handle.net/21.15107/rcub_intor_90 .